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Xu, Haodong
One or more keywords matched the following items that are connected to
Xu, Haodong
Item Type
Name
Concept
Myocytes, Cardiac
Academic Article
Elevated MCU Expression by CaMKIIdB Limits Pathological Cardiac Remodeling.
Academic Article
A small-molecule LF3 abrogates ß-catenin/TCF4-mediated suppression of NaV1.5 expression in HL-1 cardiomyocytes.
Academic Article
Enhancement of ß-catenin/T-cell factor 4 signaling causes susceptibility to cardiac arrhythmia by suppressing NaV1.5 expression in mice.
Academic Article
Opposing roles of TCF7/LEF1 and TCF7L2 in cyclin D2 and Bmp4 expression and cardiomyocyte cell cycle control during late heart development.
Academic Article
Activation of Wnt/ß-catenin signaling by hydrogen peroxide transcriptionally inhibits NaV1.5 expression.
Academic Article
APC controls asymmetric Wnt/ß-catenin signaling and cardiomyocyte proliferation gradient in the heart.
Academic Article
Dynamic monitoring of oxidative DNA double-strand break and repair in cardiomyocytes.
Academic Article
Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and ß3 subunit.
Academic Article
Activation of extracellular signal-regulated kinase 5 reduces cardiac apoptosis and dysfunction via inhibition of a phosphodiesterase 3A/inducible cAMP early repressor feedback loop.
Academic Article
Role of Ca2+/calmodulin-stimulated cyclic nucleotide phosphodiesterase 1 in mediating cardiomyocyte hypertrophy.
Academic Article
Reactive oxygen species-induced activation of p90 ribosomal S6 kinase prolongs cardiac repolarization through inhibiting outward K+ channel activity.
Academic Article
Fatty Acids Enhance the Maturation of Cardiomyocytes Derived from Human Pluripotent Stem Cells.
Academic Article
Targeted deletion of Kv4.2 eliminates I(to,f) and results in electrical and molecular remodeling, with no evidence of ventricular hypertrophy or myocardial dysfunction.
Academic Article
Reactive oxygen species suppress cardiac NaV1.5 expression through Foxo1.
Academic Article
Transgenic expression of fatty acid transport protein 1 in the heart causes lipotoxic cardiomyopathy.
Academic Article
Propofol Provides Cardiac Protection by Suppressing the Proteasome Degradation of Caveolin-3 in Ischemic/Reperfused Rat Hearts.
Academic Article
A positive feedback loop of phosphodiesterase 3 (PDE3) and inducible cAMP early repressor (ICER) leads to cardiomyocyte apoptosis.
Search Criteria
Myocytes Cardiac