B. Scott Segal to Disease Models, Animal
This is a "connection" page, showing publications B. Scott Segal has written about Disease Models, Animal.
Connection Strength
0.319
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Stoolman JS, Duncker PC, Huber AK, Giles DA, Washnock-Schmid JM, Soulika AM, Segal BM. An IFN?/CXCL2 regulatory pathway determines lesion localization during EAE. J Neuroinflammation. 2018 Jul 16; 15(1):208.
Score: 0.119
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Segal BM. Stage-specific immune dysregulation in multiple sclerosis. J Interferon Cytokine Res. 2014 Aug; 34(8):633-40.
Score: 0.090
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Kalinski AL, Yoon C, Huffman LD, Duncker PC, Kohen R, Passino R, Hafner H, Johnson C, Kawaguchi R, Carbajal KS, Jara JS, Hollis E, Geschwind DH, Segal BM, Giger RJ. Analysis of the immune response to sciatic nerve injury identifies efferocytosis as a key mechanism of nerve debridement. Elife. 2020 12 02; 9.
Score: 0.035
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Neal LM, Xing E, Xu J, Kolbe JL, Osterholzer JJ, Segal BM, Williamson PR, Olszewski MA. CD4+ T Cells Orchestrate Lethal Immune Pathology despite Fungal Clearance during Cryptococcus neoformans Meningoencephalitis. mBio. 2017 11 21; 8(6).
Score: 0.028
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Cravens PD, Hussain RZ, Miller-Little WA, Ben LH, Segal BM, Herndon E, Stüve O. IL-12/IL-23p40 Is Highly Expressed in Secondary Lymphoid Organs and the CNS during All Stages of EAE, but Its Deletion Does Not Affect Disease Perpetuation. PLoS One. 2016; 11(10):e0165248.
Score: 0.026
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Rainey-Barger EK, Blakely PK, Huber AK, Segal BM, Irani DN. Virus-induced CD8+ T cells accelerate the onset of experimental autoimmune encephalomyelitis: implications for how viral infections might trigger multiple sclerosis exacerbations. J Neuroimmunol. 2013 Jun 15; 259(1-2):47-54.
Score: 0.021