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Myocardial Hypertrophy and Compensatory Increase in Systolic Function in a Mouse Model of Oxidative Stress.
Cancer cachexia in a mouse model of oxidative stress.
Using MRI to measure in vivo free radical production and perfusion dynamics in a mouse model of elevated oxidative stress and neurogenic atrophy.
Neuroprotective effects of PPARa in retinopathy of type 1 diabetes.
Mitochondrial oxidative stress impairs contractile function but paradoxically increases muscle mass via fibre branching.
Oxidative stress-induced dysregulation of excitation-contraction coupling contributes to muscle weakness.
Nrf2 deficiency exacerbates age-related contractile dysfunction and loss of skeletal muscle mass.
Redox control of skeletal muscle atrophy.
Pharmacological targeting of mitochondrial reactive oxygen species counteracts diaphragm weakness in chronic heart failure.
NAD(P)H oxidase subunit p47phox is elevated, and p47phox knockout prevents diaphragm contractile dysfunction in heart failure.
Diaphragm dysfunction caused by sphingomyelinase requires the p47(phox) subunit of NADPH oxidase.