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Encephalomyelitis, Autoimmune, Experimental
PRMT1 Plays a Critical Role in Th17 Differentiation by Regulating Reciprocal Recruitment of STAT3 and STAT5.
A two-amino-acid substitution in the transcription factor ROR?t disrupts its function in TH17 differentiation but not in thymocyte development.
TCF-1 Inhibits IL-17 Gene Expression To Restrain Th17 Immunity in a Stage-Specific Manner.
SRC3 Is a Cofactor for ROR?t in Th17 Differentiation but Not Thymocyte Development.
Sumoylation of ROR?t regulates TH17 differentiation and thymocyte development.
Encephalomyelitis Autoimmune Experimental