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Molecular mechanisms and biological significance of CTL avidity.
Cutting edge: Dendritic cells prime a high avidity CTL response independent of the level of presented antigen.
High avidity cytotoxic T lymphocytes to a foreign antigen are efficiently activated following immunization with a recombinant paramyxovirus, simian virus 5.
Optimal colocalization of TCR and CD8 as a novel mechanism for the control of functional avidity.
Dose-dependent modulation of CD8 and functional avidity as a result of peptide encounter.
Increased sensitivity to antigen in high avidity CD8(+) T cells results from augmented membrane proximal T-cell receptor signal transduction.
In vivo modulation of avidity in highly sensitive CD8(+) effector T cells following viral infection.
Correlation between CD8 dependency and determinant density using peptide-induced, Ld-restricted cytotoxic T lymphocytes.
Control of cytotoxic T cell differentiation and activity
T Lymphocytes Cytotoxic