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Inhibition of histone deacetylation enhances endotoxin-stimulated transcription but does not reverse endotoxin tolerance.
Regulation of IL-1 receptor-associated kinases by lipopolysaccharide.
MicroRNAs distinguish translational from transcriptional silencing during endotoxin tolerance.
Mitogen-activated protein kinase phosphatase 1 disrupts proinflammatory protein synthesis in endotoxin-adapted monocytes.
GAPDH Binding to TNF-a mRNA Contributes to Posttranscriptional Repression in Monocytes: A Novel Mechanism of Communication between Inflammation and Metabolism.
Epigenetics of Severe Systemic Inflammation
FUNCTIONAL AND METABOLIC PROPERTIES OF TOXIC NEUTROPHILS