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Macrophage ABCA1 reduces MyD88-dependent Toll-like receptor trafficking to lipid rafts by reduction of lipid raft cholesterol.
The Nlrp3 inflammasome promotes age-related thymic demise and immunosenescence.
Proteomic Analysis of ABCA1-Null Macrophages Reveals a Role for Stomatin-Like Protein-2 in Raft Composition and Toll-Like Receptor Signaling.
Hepatocyte ABCA1 Deletion Impairs Liver Insulin Signaling and Lipogenesis.