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Principals of neovascularization for tissue engineering.
VEGF165 mediates formation of complexes containing VEGFR-2 and neuropilin-1 that enhance VEGF165-receptor binding.
In vivo administration of vascular endothelial growth factor (VEGF) and its antagonist, soluble neuropilin-1, predicts a role of VEGF in the progression of acute myeloid leukemia in vivo.
Pancreatic tumor growth is regulated by the balance between positive and negative modulators of angiogenesis.