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One or more keywords matched the following items that are connected to McCall, Charles
Item TypeName
Academic Article Induction of RelB participates in endotoxin tolerance.
Academic Article Assembly of inflammation-related genes for pathway-focused genetic analysis.
Academic Article G9a and HP1 couple histone and DNA methylation to TNFalpha transcription silencing during endotoxin tolerance.
Academic Article Chromatin-specific remodeling by HMGB1 and linker histone H1 silences proinflammatory genes during endotoxin tolerance.
Academic Article The NF-kappaB factor RelB and histone H3 lysine methyltransferase G9a directly interact to generate epigenetic silencing in endotoxin tolerance.
Academic Article Adoptive transfer of CD34(+) cells during murine sepsis rebalances macrophage lipopolysaccharide responses.
Academic Article The pathogenesis of pulmonary contusion: an open chest model in the rat.
Academic Article Gene silencing in severe systemic inflammation.
Academic Article Endogenous signals released from necrotic cells augment inflammatory responses to bacterial endotoxin.
Academic Article MicroRNAs distinguish translational from transcriptional silencing during endotoxin tolerance.
Academic Article Gene-specific epigenetic regulation in serious infections with systemic inflammation.
Academic Article Epigenetics, bioenergetics, and microRNA coordinate gene-specific reprogramming during acute systemic inflammation.
Academic Article Adiponectin-deficiency exaggerates sepsis-induced microvascular dysfunction in the mouse brain.
Academic Article Myeloid-derived suppressor cells evolve during sepsis and can enhance or attenuate the systemic inflammatory response.
Academic Article Fueling the flame: bioenergy couples metabolism and inflammation.
Concept Inflammation
Concept Inflammation Mediators
Academic Article MicroRNA-146a and RBM4 form a negative feed-forward loop that disrupts cytokine mRNA translation following TLR4 responses in human THP-1 monocytes.
Academic Article Deacetylation by SIRT1 Reprograms Inflammation and Cancer.
Academic Article Complement mediates a primed inflammatory response after traumatic lung injury.
Academic Article SIRT1 inhibition during the hypoinflammatory phenotype of sepsis enhances immunity and improves outcome.
Academic Article Epigenetic coordination of acute systemic inflammation: potential therapeutic targets.
Academic Article Sequential actions of SIRT1-RELB-SIRT3 coordinate nuclear-mitochondrial communication during immunometabolic adaptation to acute inflammation and sepsis.
Academic Article Resveratrol attenuates microvascular inflammation in sepsis via SIRT-1-Induced modulation of adhesion molecules in ob/ob mice.
Academic Article GAPDH Binding to TNF-a mRNA Contributes to Posttranscriptional Repression in Monocytes: A Novel Mechanism of Communication between Inflammation and Metabolism.
Academic Article Sirtuins Link Inflammation and Metabolism.
Academic Article Sirtuin-2 Regulates Sepsis Inflammation in ob/ob Mice.
Academic Article Correction: Sirtuin-2 Regulates Sepsis Inflammation in ob/ob Mice.
Grant NF-kB and Chromatin Changes in Human Sepsis
Grant Epigenetics of Severe Systemic Inflammation
Grant Epigenetics of Severe Systemic Inflammation
Grant Mitochondrial Biogenesis is Regulated by RelB During Inflammation
Academic Article Frontline Science: Myeloid cell-specific deletion of Cebpb decreases sepsis-induced immunosuppression in mice.
Academic Article Sirtuin 2 Regulates Microvascular Inflammation during Sepsis.
Grant Metabolic and Immune Properties of Sepsis
Grant Redox control over metabolism and mitochondrial bioenergetics directs the course of acute inflammation and sepsis.
Academic Article Sirtuins and Immuno-Metabolism of Sepsis.
Academic Article Frontline Science: Monocytes sequentially rewire metabolism and bioenergetics during an acute inflammatory response.
Academic Article Epigenetic and metabolic programming of innate immunity in sepsis.
Academic Article Cysteine thiol oxidation on SIRT2 regulates inflammation in obese mice with sepsis.
Academic Article S100A9 maintains myeloid-derived suppressor cells in chronic sepsis by inducing miR-21 and miR-181b.
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  • Inflammation