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Match	Type	Why
Histone-Lysine N-Methyltransferase	Concept	Why?
A functional single nucleotide polymorphism of SET8 is prognostic for breast cancer.	Academic Article	Why?
Coordinated histone modifications mediated by a CtBP co-repressor complex.	Academic Article	Why?
Enteropathy-associated T cell lymphoma subtypes are characterized by loss of function of SETD2.	Academic Article	Why?
MiR-502/SET8 regulatory circuit in pathobiology of breast cancer.	Academic Article	Why?
A crucial RNA-binding lysine residue in the Nab3 RRM domain undergoes SET1 and SET3-responsive methylation.	Academic Article	Why?
G9a and HP1 couple histone and DNA methylation to TNFalpha transcription silencing during endotoxin tolerance.	Academic Article	Why?
Proteomic and genomic approaches reveal critical functions of H3K9 methylation and heterochromatin protein-1? in reprogramming to pluripotency.	Academic Article	Why?
The Genetic Basis of Hepatosplenic T-cell Lymphoma.	Academic Article	Why?
The NF-kappaB factor RelB and histone H3 lysine methyltransferase G9a directly interact to generate epigenetic silencing in endotoxin tolerance.	Academic Article	Why?
McCall, Charles	Person	Why?
Human LSD2/KDM1b/AOF1 regulates gene transcription by modulating intragenic H3K4me2 methylation.	Academic Article	Why?
Re: AKT-Mediated Stabilization of Histone Methyltransferase WHSC1 Promotes Prostate Cancer Metastasis.	Academic Article	Why?
Zhang, Wei	Person	Why?
Dynamic and selective nucleosome repositioning during endotoxin tolerance.	Academic Article	Why?

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