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Connection

B. Scott Segal to Encephalomyelitis, Autoimmune, Experimental

This is a "connection" page, showing publications B. Scott Segal has written about Encephalomyelitis, Autoimmune, Experimental.
  1. Stoolman JS, Duncker PC, Huber AK, Giles DA, Washnock-Schmid JM, Soulika AM, Segal BM. An IFN?/CXCL2 regulatory pathway determines lesion localization during EAE. J Neuroinflammation. 2018 Jul 16; 15(1):208.
    View in: PubMed
    Score: 0.614
  2. Duncker PC, Stoolman JS, Huber AK, Segal BM. GM-CSF Promotes Chronic Disability in Experimental Autoimmune Encephalomyelitis by Altering the Composition of Central Nervous System-Infiltrating Cells, but Is Dispensable for Disease Induction. J Immunol. 2018 02 01; 200(3):966-973.
    View in: PubMed
    Score: 0.591
  3. Grifka-Walk HM, Segal BM. T-bet promotes the accumulation of encephalitogenic Th17 cells in the CNS. J Neuroimmunol. 2017 03 15; 304:35-39.
    View in: PubMed
    Score: 0.529
  4. Grifka-Walk HM, Giles DA, Segal BM. IL-12-polarized Th1 cells produce GM-CSF and induce EAE independent of IL-23. Eur J Immunol. 2015 Oct; 45(10):2780-6.
    View in: PubMed
    Score: 0.501
  5. Carbajal KS, Mironova Y, Ulrich-Lewis JT, Kulkarni D, Grifka-Walk HM, Huber AK, Shrager P, Giger RJ, Segal BM. Th Cell Diversity in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis. J Immunol. 2015 Sep 15; 195(6):2552-9.
    View in: PubMed
    Score: 0.500
  6. Rumble JM, Huber AK, Krishnamoorthy G, Srinivasan A, Giles DA, Zhang X, Wang L, Segal BM. Neutrophil-related factors as biomarkers in EAE and MS. J Exp Med. 2015 Jan 12; 212(1):23-35.
    View in: PubMed
    Score: 0.481
  7. Segal BM. Stage-specific immune dysregulation in multiple sclerosis. J Interferon Cytokine Res. 2014 Aug; 34(8):633-40.
    View in: PubMed
    Score: 0.467
  8. Stoolman JS, Duncker PC, Huber AK, Segal BM. Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitis. J Immunol. 2014 Jul 15; 193(2):564-70.
    View in: PubMed
    Score: 0.462
  9. Grifka-Walk HM, Lalor SJ, Segal BM. Highly polarized Th17 cells induce EAE via a T-bet independent mechanism. Eur J Immunol. 2013 Nov; 43(11):2824-31.
    View in: PubMed
    Score: 0.437
  10. Lalor SJ, Segal BM. Th1-mediated experimental autoimmune encephalomyelitis is CXCR3 independent. Eur J Immunol. 2013 Nov; 43(11):2866-74.
    View in: PubMed
    Score: 0.437
  11. Segal BM. The unwavering commitment of regulatory T cells in the suppression of autoimmune encephalomyelitis: another aspect of immune privilege in the CNS. Eur J Immunol. 2012 May; 42(5):1102-5.
    View in: PubMed
    Score: 0.399
  12. Rao P, Segal BM. Experimental autoimmune encephalomyelitis. Methods Mol Biol. 2012; 900:363-80.
    View in: PubMed
    Score: 0.390
  13. Kroenke MA, Segal BM. IL-23 modulated myelin-specific T cells induce EAE via an IFN? driven, IL-17 independent pathway. Brain Behav Immun. 2011 Jul; 25(5):932-7.
    View in: PubMed
    Score: 0.359
  14. Kroenke MA, Chensue SW, Segal BM. EAE mediated by a non-IFN-?/non-IL-17 pathway. Eur J Immunol. 2010 Aug; 40(8):2340-8.
    View in: PubMed
    Score: 0.354
  15. Segal BM. Th17 cells in autoimmune demyelinating disease. Semin Immunopathol. 2010 Mar; 32(1):71-7.
    View in: PubMed
    Score: 0.343
  16. Kroenke MA, Carlson TJ, Andjelkovic AV, Segal BM. IL-12- and IL-23-modulated T cells induce distinct types of EAE based on histology, CNS chemokine profile, and response to cytokine inhibition. J Exp Med. 2008 Jul 07; 205(7):1535-41.
    View in: PubMed
    Score: 0.306
  17. Segal BM. The role of natural killer cells in curbing neuroinflammation. J Neuroimmunol. 2007 Nov; 191(1-2):2-7.
    View in: PubMed
    Score: 0.290
  18. Kroenke MA, Segal BM. Th17 and Th1 responses directed against the immunizing epitope, as opposed to secondary epitopes, dominate the autoimmune repertoire during relapses of experimental autoimmune encephalomyelitis. J Neurosci Res. 2007 Jun; 85(8):1685-93.
    View in: PubMed
    Score: 0.284
  19. Giles DA, Washnock-Schmid JM, Duncker PC, Dahlawi S, Ponath G, Pitt D, Segal BM. Myeloid cell plasticity in the evolution of central nervous system autoimmunity. Ann Neurol. 2018 01; 83(1):131-141.
    View in: PubMed
    Score: 0.148
  20. Cravens PD, Hussain RZ, Miller-Little WA, Ben LH, Segal BM, Herndon E, Stüve O. IL-12/IL-23p40 Is Highly Expressed in Secondary Lymphoid Organs and the CNS during All Stages of EAE, but Its Deletion Does Not Affect Disease Perpetuation. PLoS One. 2016; 11(10):e0165248.
    View in: PubMed
    Score: 0.136
  21. Sandy AR, Stoolman J, Malott K, Pongtornpipat P, Segal BM, Maillard I. Notch signaling regulates T cell accumulation and function in the central nervous system during experimental autoimmune encephalomyelitis. J Immunol. 2013 Aug 15; 191(4):1606-13.
    View in: PubMed
    Score: 0.108
  22. Rainey-Barger EK, Blakely PK, Huber AK, Segal BM, Irani DN. Virus-induced CD8+ T cells accelerate the onset of experimental autoimmune encephalomyelitis: implications for how viral infections might trigger multiple sclerosis exacerbations. J Neuroimmunol. 2013 Jun 15; 259(1-2):47-54.
    View in: PubMed
    Score: 0.107
  23. Rainey-Barger EK, Rumble JM, Lalor SJ, Esen N, Segal BM, Irani DN. The lymphoid chemokine, CXCL13, is dispensable for the initial recruitment of B cells to the acutely inflamed central nervous system. Brain Behav Immun. 2011 Jul; 25(5):922-31.
    View in: PubMed
    Score: 0.090
  24. Segal BM. Getting to the crux of the matter: IL-23 and Th17 cell accumulation in the CNS. Eur J Immunol. 2009 Jul; 39(7):1713-5.
    View in: PubMed
    Score: 0.082
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.