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The antinociceptive effects of spinal cyclooxygenase inhibitors on uterine cervical distension.
Chronic estrogen sensitizes a subset of mechanosensitive afferents innervating the uterine cervix.
Uterine cervical afferents in thoracolumbar dorsal root ganglia express transient receptor potential vanilloid type 1 channel and calcitonin gene-related peptide, but not P2X3 receptor and somatostatin.
Estrogen amplifies pain responses to uterine cervical distension in rats by altering transient receptor potential-1 function.
Pregnancy increases excitability of mechanosensitive afferents innervating the uterine cervix.
Neurophysiology of Pain from the Uterine Cervix