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MicroRNA 21 (miR-21) and miR-181b couple with NFI-A to generate myeloid-derived suppressor cells and promote immunosuppression in late sepsis.
Epigenetics of Severe Systemic Inflammation
FUNCTIONAL AND METABOLIC PROPERTIES OF TOXIC NEUTROPHILS
Mitochondrial Biogenesis is Regulated by RelB During Inflammation
Frontline Science: Myeloid cell-specific deletion of Cebpb decreases sepsis-induced immunosuppression in mice.
Expression of C/EBPß in myeloid progenitors during sepsis promotes immunosuppression.
IL-10 induces an immune repressor pathway in sepsis by promoting S100A9 nuclear localization and MDSC development.
S100A9 maintains myeloid-derived suppressor cells in chronic sepsis by inducing miR-21 and miR-181b.