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One or more keywords matched the following items that are connected to Ferrario, Carlos
Item TypeName
Academic Article Angiotensin-(1-7) inhibits growth of cardiac myocytes through activation of the mas receptor.
Academic Article Mineralocorticoid receptor blockade attenuates chronic overexpression of the renin-angiotensin-aldosterone system stimulation of reduced nicotinamide adenine dinucleotide phosphate oxidase and cardiac remodeling.
Academic Article Localization of the novel angiotensin peptide, angiotensin-(1-12), in heart and kidney of hypertensive and normotensive rats.
Academic Article Regulation of ACE2 in cardiac myocytes and fibroblasts.
Academic Article Uptake and metabolism of the novel peptide angiotensin-(1-12) by neonatal cardiac myocytes.
Academic Article Chymase-dependent generation of angiotensin II from angiotensin-(1-12) in human atrial tissue.
Academic Article Cardiac angiotensin-(1-7) in ischemic cardiomyopathy.
Concept Myocytes, Cardiac
Academic Article Renin inhibition and AT(1)R blockade improve metabolic signaling, oxidant stress and myocardial tissue remodeling.
Academic Article Chymase mediates injury and mitochondrial damage in cardiomyocytes during acute ischemia/reperfusion in the dog.
Academic Article Modulation of cardiac L-type Ca2+ current by angiotensin-(1-7): normal versus heart failure.
Academic Article Cardiomyocyte-specific deletion of the G protein-coupled estrogen receptor (GPER) leads to left ventricular dysfunction and adverse remodeling: A sex-specific gene profiling analysis.
Academic Article Cellular basis of angiotensin-(1-7)-induced augmentation of left ventricular functional performance in heart failure.
Academic Article Inflammatory and mitochondrial gene expression data in GPER-deficient cardiomyocytes from male and female mice.
Grant Vaso-Hormonal Mechanisms in Hypertension
Academic Article Blunting of estrogen modulation of cardiac cellular chymase/RAS activity and function in SHR.
Academic Article Critical role of the chymase/angiotensin-(1-12) axis in modulating cardiomyocyte contractility.
Academic Article Estrogen modulates the differential expression of cardiac myocyte chymase isoforms and diastolic function.
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  • Myocytes Cardiac